Bisphenol A in chronic kidney disease
Entidad
UAM. Departamento de MedicinaEditor
HindawiFecha de edición
2013-09-23Cita
10.1155/2013/437857
International Journal of Nephrology 2013 (2013): 437857
ISSN
2090-214XDOI
10.1155/2013/437857Financiado por
This research was supported by a grant from ISCIII and FEDER funds PS09/00447, Sociedad Española de Nefrologia, ISCIII-RETICREDinREN/RD06/0016, RD12/0021/,Comunidad de Madrid/CIFRA/S2010/BMD-2378, and salary support was from ERA/EDTA to Usama Elewa and Programa Intensificación Actividad Investigadora (ISCIII/Agencia Laín-Entralgo/CM) to Alberto Ortiz Arduán. ISC III (PI10/ 00072), RECAVA (RD06/0014/0038) and Lilly Foundation to Jesús Egido.Proyecto
Comunidad de Madrid. S2010/BMD-2378/CIFRAVersión del editor
http://dx.doi.org/10.1155/2013/437857Materias
Kidney; Bisphenol A; MedicinaDerechos
© 2013 Emilio González-Parra et al.Resumen
Phenols are uremic toxins of intestinal origin formed by bacteria during protein metabolism. Of these molecules, p-cresol is the most studied and has been associated with renal function impairment and vascular damage. Bisphenol A (BPA) is a molecule with structural similarity with phenols found in plastic food and beverage containers as well as in some dialyzers. BPA is considered an environmental toxicant based on animal and cell culture studies. Japanese authorities recently banned BPA use in baby bottles based on observational association studies in newborns. BPA is excreted in urine and uremic patients present higher serum levels, but there is insufficient evidence to set cut-off levels or to link BPA to any harmful effect in CKD. However, the renal elimination and potential exposure during dialysis warrant the monitoring of BPA exposure and the design of observational studies in which the potential health risks of BPA for end-stage renal disease patients are evaluated.
Lista de ficheros
Google Scholar:González-Parra, Emilio
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Herrero, José Antonio
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Elewa, Usama
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Bosch, Ricardo J.
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Ortiz Arduán, Alberto
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Egido, Jesús
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